Bifidobacterium ⇒ Bifidobacterium pseudocatenulatum {10000127}

Shared Reference Notes

• [1.1] [#Bacteroides cellulosilyticus] [#Human milk oligosaccharides] [#Human breast milk, #Infants] 
  - The relative abundance of maternal B. cellulosilyticus, a versatile carbohydrate degrader, was positively associated with the overall abundance of microbial glycoside hydrolases in the infant gut, particularly those involved in HMO degradation. - Moreover, this maternal species was inversely correlated with intact HMOs in infant fecal samples and positively associated with HMO-utilizing infant species that are unable to degrade these oligosaccharides, such as Bifidobacterium pseudocatenulatum.
• [1.2] [#Rheumatoid Arthritis] [#Secondary bile acids] [#Probiotic Bifidobacterium pseudocatenulatum] 
  - In a collagen-induced arthritis (CIA) mouse model, B. pseudocatenulatum prevented joint damage by protecting the intestinal barrier and reshaped gut microbial composition, thereby elevating bile salt hydrolase (BSH) enzyme activity and increasing the levels of unconjugated secondary BAs to suppress aberrant T-helper 1/17-type immune responses
• [1.3] [#Bifidobacteria, #Bifidobacterium adolescentis] [#Fructooligosaccharides, #Human milk oligosaccharides, #Inulin] 
  - 2′FL/LNnT were bifidogenic for both age groups, 3′SL/6′SL and FOS/IN were exclusively bifidogenic for children and adults, respectively. - 3′SL/6′SL stimulated B. pseudocatenulatum (abundant in children), FOS/IN enhanced B. adolescentis (abundant in adults). - increased #Acetate, #Propionate and #Butyrate (only in adults) with product- and age-dependent differences.
• [1.4] [#Infants] 
  - B. pseudocatenulatum > commonly associated with weaning to solid food in early age
• [1.5] [#Gastric carcinoma, #Peptic ulcer] [#Bifidobacterium adolescentis, #Bifidobacterium bifidum, #Bifidobacterium breve, #Bifidobacterium longum, #Chloracidobacterium thermophilum B, #Helicobacter pylori, #Limosilactobacillus (Lactobacillus) reuteri] 
  - H. pylori infection alone is not sufficient to develop severe gastric abnormalities such as gastric cancer and gastric ulcers. - A lower abundance of gut resident #Bifidobacterium genus with specific strains ( B. adolescentis, B. bifidum, B. breve, B. longum, B. moukalabense, B. pseudocatenulatum, and B. reuteri.) plays a significant role in developing severe gastric malignancy.
• [1.6] 
  - In #Gout, #Bacteroides caccae and #Bacteroides xylanisolvens are enriched. - #Faecalibacterium prausnitzii and Bifidobacterium pseudocatenulatum depleted. - The established reference microbial gene catalogue for #Gout revealed disorder in purine degradation and #Butyric acid biosynthesis in #Gout patients. - Intestinal microbiota of #Gout are more similar to those of type-2 diabetes than to liver cirrhosis, whereas depletion of #Faecalibacterium prausnitzii and reduced butyrate biosynthesis are shared in each of the metabolic syndromes. - #Bacteroides caccae is one biomarker of IBD. OmpW protein produced by B. caccae is a target of the IBD-associated immune response, thus the enriched intestinal B. caccae in #Gout patients could potentially induce serious inflammatory response. - #Faecalibacterium prausnitzii has anti-inflammatory properties and contribute to gut health through butyrate production. for #Gout patients, potentially explaining the decline in #Butyric acid biosynthesis in #Gout patients.

References Notes